Paper
1 May 2007 Cellular rehabilitation of photobiomodulation
Timon Cheng-Yi Liu, Jian-Qin Yuan, Yan-Fang Wang, Xiao-Yang Xu, Song-Hao Liu
Author Affiliations +
Proceedings Volume 6534, Fifth International Conference on Photonics and Imaging in Biology and Medicine; 653403 (2007) https://doi.org/10.1117/12.741093
Event: Fifth International Conference on Photonics and Imaging in Biology and Medicine, 2006, Wuhan, China
Abstract
Homeostasis is a term that refers to constancy in a system. A cell in homeostasis normally functions. There are two kinds of processes in the internal environment and external environment of a cell, the pathogenic processes (PP) which disrupts the old homeostasis (OH), and the sanogenetic processes (SP) which restores OH or establishes a new homeostasis (NH). Photobiomodualtion (PBM), the cell-specific effects of low intensity monochromatic light or low intensity laser irradiation (LIL) on biological systems, is a kind of modulation on PP or SP so that there is no PBM on a cell in homeostasis. There are two kinds of pathways mediating PBM, the membrane endogenetic chromophores mediating pathways which often act through reactive oxygen species, and membrane proteins mediating pathways which often enhance cellular SP so that it might be called cellular rehabilitation. The cellular rehabilitation of PBM will be discussed in this paper. It is concluded that PBM might modulate the disruption of cellular homeostasis induced by pathogenic factors such as toxin until OH has been restored or NH has been established, but can not change homeostatic processes from one to another one.
© (2007) COPYRIGHT Society of Photo-Optical Instrumentation Engineers (SPIE). Downloading of the abstract is permitted for personal use only.
Timon Cheng-Yi Liu, Jian-Qin Yuan, Yan-Fang Wang, Xiao-Yang Xu, and Song-Hao Liu "Cellular rehabilitation of photobiomodulation", Proc. SPIE 6534, Fifth International Conference on Photonics and Imaging in Biology and Medicine, 653403 (1 May 2007); https://doi.org/10.1117/12.741093
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Cited by 2 scholarly publications.
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KEYWORDS
Surface plasmons

Pathogens

Modulation

Blood

Proteins

Cell death

Chromophores

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